Long-term follow-Up of an infant with thyrotoxicosis due to germline mutation of the TSH receptor gene (Met453Thr)

Horm Res. 1999;51(1):43-6. doi: 10.1159/000023312.

Abstract

A 18-year clinical follow-up period in a male patient with a germline TSH-R gene mutation (Met453Thr) is described. Nonautoimmune thyrotoxicosis was diagnosed at the age of 7 months. The patient had exophthalmus, failure to thrive, advanced bone age and no goiter. Long-term antithyroid drug treatment (ATD) was necessary during childhood. At the age of 7 years he developed a goiter. Subtotal thyroidectomy was performed at the age of 9 years, followed by repeated ablative radiotherapy at the age of 9.5-13 years due to a toxic multinodular goiter. After 13 years ATD could be discontinued and the patient was euthyroid until 16 years of age, where L-thyroxine substitution had to be started. The exophthalmus diminished, and had disappeared at the age of 18 years, when CT scan of the orbit was performed.

Conclusion: TSH-R mutation must be considered in early nonautoimmune thyrotoxicosis. A very aggressive treatment strategy is necessary.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Substitution*
  • Exophthalmos / therapy
  • Follow-Up Studies
  • Germ-Line Mutation*
  • Goiter, Nodular / drug therapy
  • Goiter, Nodular / etiology
  • Goiter, Nodular / radiotherapy
  • Growth
  • Humans
  • Infant
  • Male
  • Methionine
  • Receptors, Thyrotropin / genetics*
  • Threonine
  • Thyroidectomy
  • Thyrotoxicosis / genetics*
  • Thyrotoxicosis / physiopathology*
  • Thyrotoxicosis / therapy
  • Thyroxine / therapeutic use
  • Time Factors

Substances

  • Receptors, Thyrotropin
  • Threonine
  • Methionine
  • Thyroxine