ret/PTC expression may be associated with local invasion of thyroid papillary carcinoma

J Surg Oncol. 1999 Jun;71(2):76-81; discussion 81-2. doi: 10.1002/(sici)1096-9098(199906)71:2<76::aid-jso4>3.0.co;2-0.

Abstract

Background and objectives: The exact role of ret/PTC in the development of papillary carcinoma remains unclear. Expression of the ret/PTC oncogene was examined immunohistochemically to address its role in the progression of thyroid carcinomas.

Methods: Paraffin-embedded samples from 34 clinically evident thyroid papillary carcinomas and 19 occult papillary carcinomas were analyzed using an antibody raised against the ret tyrosine kinase domain.

Results: Expression of ret/PTC was demonstrated in 6/19 (32%) occult carcinomas. The frequency of expression of ret/PTC in clinically evident carcinomas in 16/34 (47%) was significantly higher than in normal tissues (0%) and follicular adenomas (1/14, 7%, P < 0.01).ret/PTC expression was observed more frequently in the peripheral areas of clinically evident carcinomas (P < 0.01). Although there was no correlation of ret/PTC expression with tumor size, lymph node metastasis, or distant metastasis, the incidence of ret/PTC expression in tumors with extrathyroidal invasion (13/19, 68%) was significantly higher than those without extrathyroidal invasion (3/15, 20%, P < 0.01). Local invasion was found in none of the occult carcinomas. The frequency of expression in occult carcinomas was significantly lower than in clinically evident carcinomas with extrathyroidal invasion (P < 0.05).

Conclusions: The ret/PTC oncogene may be involved in the local invasion of thyroid papillary carcinomas.

MeSH terms

  • Adenoma / metabolism
  • Adenoma / pathology
  • Carcinoma, Medullary / metabolism
  • Carcinoma, Medullary / pathology
  • Carcinoma, Medullary / secondary
  • Carcinoma, Papillary / metabolism*
  • Carcinoma, Papillary / pathology*
  • Carcinoma, Papillary / secondary
  • Humans
  • Immunohistochemistry
  • Lymphatic Metastasis
  • Neoplasm Invasiveness
  • Nuclear Receptor Coactivators
  • Oncogene Proteins / biosynthesis*
  • Thyroid Neoplasms / metabolism*
  • Thyroid Neoplasms / pathology*
  • Transcription Factors*

Substances

  • NCOA4 protein, human
  • Nuclear Receptor Coactivators
  • Oncogene Proteins
  • Transcription Factors