A structural change in the kinesin motor protein that drives motility

Nature. 1999 Dec 16;402(6763):778-84. doi: 10.1038/45483.

Abstract

Kinesin motors power many motile processes by converting ATP energy into unidirectional motion along microtubules. The force-generating and enzymatic properties of conventional kinesin have been extensively studied; however, the structural basis of movement is unknown. Here we have detected and visualized a large conformational change of an approximately 15-amino-acid region (the neck linker) in kinesin using electron paramagnetic resonance, fluorescence resonance energy transfer, pre-steady state kinetics and cryo-electron microscopy. This region becomes immobilized and extended towards the microtubule 'plus' end when kinesin binds microtubules and ATP, and reverts to a more mobile conformation when gamma-phosphate is released after nucleotide hydrolysis. This conformational change explains both the direction of kinesin motion and processive movement by the kinesin dimer.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenosine Triphosphatases / metabolism
  • Cloning, Molecular
  • Cryoelectron Microscopy
  • Cysteine / chemistry
  • Cysteine / physiology
  • Humans
  • Kinesins / chemistry
  • Kinesins / physiology*
  • Microtubules / physiology
  • Models, Molecular
  • Molecular Motor Proteins / chemistry
  • Molecular Motor Proteins / physiology*
  • Mutagenesis
  • Protein Conformation
  • Recombinant Proteins / chemistry
  • Spectrometry, Fluorescence / methods

Substances

  • Molecular Motor Proteins
  • Recombinant Proteins
  • Adenosine Triphosphatases
  • Kinesins
  • Cysteine