Ganciclovir-sensitive acute graft-versus-host disease in mice receiving herpes simplex virus-thymidine kinase-expressing donor T cells in a bone marrow transplantation setting

Transplantation. 2000 Feb 27;69(4):503-8. doi: 10.1097/00007890-200002270-00007.

Abstract

Background: The use of donor T cells expressing the herpes simplex thymidine kinase (HSV-TK) gene followed by ganciclovir (GCV) treatment could allow for specific modulation of the alloreactivity occurring after bone marrow transplantation. We are presently exploring such an approach in a phase I clinical trial.

Methods: To examine the beneficial effect of administrating HSV-TK-expressing donor T lymphocytes +/- GCV treatment on acute graft-versus-host disease (aGVHD) control, irradiated Balb/c or C57BL/6 mice underwent transplantation with allogeneic bone marrow cells in conjunction with CD3+ allogeneic splenocytes from transgenic mice expressing an HSV-TK transgene. GCV treatment was initiated upon the occurrence of severe aGVHD.

Results: GCV treatment resulted in a 40-60% long-term survival rate of GVHD-free recipients having received HSV-TK-expressing T cells, whereas only 0-6% of mice survived without GCV treatment. Lethal aGVHD occurred in all the control animals having received non-HSV-TK-expressing T cells, irrespective of GCV treatment.

Conclusion: Our results demonstrate that the administration of donor HSV-TK-expressing T cells to hematopoietic stem cell graft recipients followed by GCV treatment at the onset of severe aGVHD significantly reduces aGVHD-induced mortality and results in GVHD-free surviving recipients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antiviral Agents / therapeutic use*
  • Bone Marrow Transplantation*
  • Ganciclovir / therapeutic use*
  • Graft vs Host Disease / mortality
  • Graft vs Host Disease / pathology*
  • Graft vs Host Disease / prevention & control*
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Mice, Transgenic
  • Simplexvirus / physiology*
  • Survival Rate
  • T-Lymphocytes / enzymology*
  • T-Lymphocytes / virology*
  • Thymidine Kinase / biosynthesis*

Substances

  • Antiviral Agents
  • Thymidine Kinase
  • Ganciclovir