Probing the catalytic mechanism of the insulin receptor kinase with a tetrafluorotyrosine-containing peptide substrate

J Biol Chem. 2000 Sep 29;275(39):30394-8. doi: 10.1074/jbc.M003524200.

Abstract

The interaction of a synthetic tetrafluorotyrosyl peptide substrate with the activated tyrosine kinase domain of the insulin receptor was studied by steady-state kinetics and x-ray crystallography. The pH-rate profiles indicate that the neutral phenol, rather than the chemically more reactive phenoxide ion, is required for enzyme-catalyzed phosphorylation. The pK(a) of the tetrafluorotyrosyl hydroxyl is elevated 2 pH units on the enzyme compared with solution, whereas the phenoxide anion species behaves as a weak competitive inhibitor of the tyrosine kinase. A structure of the binary enzyme-substrate complex shows the tetrafluorotyrosyl OH group at hydrogen bonding distances from the side chains of Asp(1132) and Arg(1136), consistent with elevation of the pK(a). These findings strongly support a reaction mechanism favoring a dissociative transition state.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Catalytic Domain*
  • Crystallography, X-Ray
  • Electrons
  • Hydrogen Bonding
  • Kinetics
  • Models, Molecular
  • Peptides / metabolism*
  • Receptor, Insulin / metabolism*
  • Tyrosine / analogs & derivatives*
  • Tyrosine / metabolism

Substances

  • Peptides
  • 2,3,5,6-tetrafluorotyrosine
  • Tyrosine
  • Receptor, Insulin