T-cell progenitor function during progressive human immunodeficiency virus-1 infection and after antiretroviral therapy

Blood. 2000 Jul 1;96(1):242-9.

Abstract

Impairment of T-cell renewal has been proposed as contributing to CD4(+) T-cell depletion in persons infected with human immunodeficiency virus-1. We analyzed the T-cell development capacity of progenitors using fetal thymus organ culture. Those who progressed to AIDS had a dramatic loss in T-cell development capacity shortly after seroconversion. In contrast, long-term nonprogressors retained progenitor capacity 8 years after seroconversion. Approximately 70% of patients experienced an improvement in T-cell development capacity after receiving 6 months of potent antiretroviral therapy. Improvement in T-cell development in fetal thymus organ culture correlated with an increase in the number of naive CD4(+) T cells in peripheral blood. Numbers of progenitors in blood and bone marrow after seroconversion or during therapy did not correlate with the change observed in T-cell development capacity. These data provide evidence that HIV-1 infection can interfere with T-cell renewal at the level of the progenitor cell. Interference with T-cell renewal may contribute to CD4(+) T-cell depletion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acquired Immunodeficiency Syndrome / immunology
  • Adult
  • Animals
  • Anti-HIV Agents / therapeutic use*
  • Bone Marrow Cells / pathology
  • Disease Progression
  • Disease-Free Survival
  • Drug Therapy, Combination
  • Follow-Up Studies
  • Genes, RAG-1
  • HIV Infections / blood
  • HIV Infections / drug therapy*
  • HIV Infections / immunology*
  • HIV Infections / pathology
  • HIV-1*
  • Hematopoietic Stem Cells / immunology*
  • Homeodomain Proteins / genetics
  • Homeodomain Proteins / physiology
  • Humans
  • Leukocytes, Mononuclear / immunology
  • Mice
  • Mice, Knockout
  • Middle Aged
  • Retrospective Studies
  • T-Lymphocytes / immunology*

Substances

  • Anti-HIV Agents
  • Homeodomain Proteins
  • RAG-1 protein