Abstract
Spontaneous voltage fluctuations often occurred during perforated-patch recordings from clustered rat carotid body (CB) chemoreceptors in vitro. This activity sometimes appeared as rhythmic-like spikes, when cluster size exceeded approximately 20 cells. Spike discharge could be augmented or induced by hypoxia or 5-HT (2-10 microM) application, and inhibited by the 5-HT receptor blocker, ketanserin (50-100 microM). Thus, endogenous 5-HT may contribute to spontaneous firing within CB receptor clusters via autocrine/paracrine mechanisms.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Action Potentials / drug effects
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Action Potentials / physiology
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Animals
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Autocrine Communication / physiology
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Carotid Body / cytology
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Carotid Body / drug effects
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Carotid Body / metabolism*
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Cell Hypoxia / physiology
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Cells, Cultured
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Chemoreceptor Cells / cytology
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Chemoreceptor Cells / drug effects
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Chemoreceptor Cells / metabolism*
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Ketanserin / pharmacology
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Paracrine Communication / physiology
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Patch-Clamp Techniques
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Periodicity*
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Rats
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Serotonin / metabolism*
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Serotonin / pharmacology
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Serotonin Antagonists / pharmacology
Substances
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Serotonin Antagonists
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Serotonin
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Ketanserin