Accumulation of copper in the liver and hepatic injury in chronic hepatitis C

J Gastroenterol Hepatol. 2000 Jul;15(7):786-91. doi: 10.1046/j.1440-1746.2000.02199.x.

Abstract

Background and aims: Relationships between chronic liver disease and trace metals have not been clearly understood. To examine connections between severity of hepatic fibrosis in chronic hepatitis C and copper, iron and zinc we measured the contents of these metals in liver tissue and serum in the patients.

Methods: Forty-one patients (26-62 years), 13 with fibrosis representing grade F1, 16 with F2, seven with F3, and five with F4, entered this study. Metals were quantified in needle liver-biopsy specimens by particle-induced X-ray emission. In serum, metals were measured by flameless atomic absorption spectrometry.

Results: Hepatic copper content increased with progression of hepatic fibrosis (P < 0.05). The copper content correlated positively with bilirubin (r = 0.466, P = 0.0023), and with type IV collagen (r = 0.402, P = 0.0086) and correlated negatively with albumin (r = -0.404, P = 0.080). However, hepatic iron and zinc contents did not show a significant differences between grades of fibrosis.

Conclusions: Copper accumulation in fibrotic livers caused by chronic hepatitis C may contribute to hepatic injury. The real mechanism is not known at present, but excess copper may damage the liver by oxidative stress.

MeSH terms

  • Adult
  • Copper / metabolism*
  • Female
  • Hepatitis C, Chronic / metabolism*
  • Humans
  • Liver Cirrhosis / metabolism*
  • Male
  • Middle Aged

Substances

  • Copper