Alpha(1)-antichymotrypsin gene polymorphism in patients with stroke

Stroke. 2000 Sep;31(9):2103-5. doi: 10.1161/01.str.31.9.2103.

Abstract

Background and purpose: A role for proteolytic enzymes has been suggested in the pathogenesis of stroke. In a search for new genetic factors, we investigated the gene polymorphism of the serine protease inhibitor alpha(1)-antichymotrypsin (ACT) in patients with stroke.

Methods: Two hundred twenty patients with acute ischemic stroke (n=182) and primary intracerebral hemorrhage (n=38) and 70 control subjects without clinical cerebrovascular disease were genotyped for the ACT polymorphism.

Results: The ACT-TT genotype was more frequent in patients with primary intracerebral hemorrhage than in patients with ischemic stroke (31.6% versus 16.4%, P:<0.05) or in control subjects (21.4%, P:=0.1). After adjusting for age, gender, and vascular risk factors, the ACT-TT genotype was associated with primary intracerebral hemorrhage, with an OR of 2.3 (95% CI 1.0 to 5. 2) compared with ischemic stroke and an OR of 1.8 (95% CI 0.85 to 9. 65) compared with controls.

Conclusions: Pending confirmation in a larger study, our results suggest that the ACT-TT genotype might be a risk factor for primary cerebral hemorrhage.

Publication types

  • Comparative Study

MeSH terms

  • Acute Disease
  • Adult
  • Aged
  • Aged, 80 and over
  • Cerebral Hemorrhage / blood
  • Cerebral Hemorrhage / diagnosis
  • Cerebral Hemorrhage / genetics*
  • Female
  • Genotype
  • Humans
  • Male
  • Middle Aged
  • Odds Ratio
  • Polymorphism, Genetic
  • Serine Proteinase Inhibitors / genetics*
  • Stroke / blood
  • Stroke / diagnosis
  • Stroke / genetics*
  • alpha 1-Antichymotrypsin / genetics*

Substances

  • Serine Proteinase Inhibitors
  • alpha 1-Antichymotrypsin