Haemophilus influenzae porin contributes to signaling of the inflammatory cascade in rat brain

Infect Immun. 2001 Jan;69(1):221-7. doi: 10.1128/IAI.69.1.221-227.2001.

Abstract

In the present study we observed that the Haemophilus influenzae type b (Hib) porin, among the different surface bacterial components, is involved in the pathophysiology of bacterial meningitis. This study demonstrates that inoculation of Hib porin into the fourth cerebral ventricle causes the simultaneous expression of interleukin-1alpha (IL-1alpha), tumor necrosis factor alpha (TNF-alpha), and macrophage inflammatory protein 2 (MIP-2) at 6 h after inoculation. At 24 h, the expression of MIP-2 decreases while the expression of IL-1alpha and TNF-alpha increases. The mRNA expression of IL-1alpha, TNF-alpha, and MIP-2 is correlated with injury to the blood-brain barrier as demonstrated by the appearance of serum proteins and leukocytes in cerebrospinal fluid and by the increase in brain water content.

MeSH terms

  • Animals
  • Brain Chemistry
  • Cytokines / genetics
  • Haemophilus influenzae / pathogenicity*
  • Male
  • Meningitis, Haemophilus / etiology*
  • Nerve Tissue Proteins / analysis
  • Porins / physiology*
  • RNA, Messenger / analysis
  • Rats
  • Rats, Sprague-Dawley

Substances

  • Cytokines
  • Nerve Tissue Proteins
  • Porins
  • RNA, Messenger