Congenital bilateral absence of the vas deferens: clinical characteristics, biological parameters, cystic fibrosis transmembrane conductance regulator gene mutations, and implications for genetic counseling

Fertil Steril. 2000 Dec;74(6):1164-74. doi: 10.1016/s0015-0282(00)01625-3.

Abstract

Objective: To evaluate relationships between the phenotypic and genotypic characteristics of patients with congenital bilateral absence of the vas deferens (CBAVD).

Design: Retrospective study.

Setting: A university hospital urology-andrology department.

Patient(s): Forty-one men with CBAVD.

Intervention(s): CBAVD was diagnosed during surgical and/or ultrasound exploration of the vasa deferentia (VD) (n = 39), or on the basis of impalpable scrotal VD (n = 2).

Main outcome measure(s): History, clinical and seminal characteristics, and cystic fibrosis transmembrane conductance regulator (CFTR) gene mutations including IVS-8 polyT analysis.

Result(s): A palpable scrotal vas deferens was present as a fibrous cord or nonpermeable duct in 13% of patients undergoing surgical exploration. Seminal vesicles were bilaterally absent in 28% of patients. No CFTR gene mutation or 5T allele was detected in 24.5% of the patients. Two CBAVD patients with renal agenesis carried a CFTR gene mutation (DeltaF508/5T-9T and R117G/7T-9T). CBAVD patients who have both a semen volume of < or =1.0 mL and a semen pH of < 7.0 have a significantly higher risk of severe CFTR gene mutation (OR = 9.12 [95% CI = 1.81-49.50]).

Conclusion(s): A palpable scrotal vas deferens was found in 13% of CBAVD patients. Semen volume of < or =1.0 mL and semen pH of < 7.0 in CBAVD patients were associated with a higher risk of severe CFTR gene mutations. Patients with CBAVD and renal agenesis should be screened for CFTR gene mutations before assisted reproductive techniques are used.

MeSH terms

  • Abnormalities, Multiple / genetics
  • Adult
  • Alleles
  • Cystic Fibrosis Transmembrane Conductance Regulator / genetics*
  • Fructose / metabolism
  • Genetic Counseling
  • Genotype
  • Humans
  • Kidney / abnormalities
  • Kidney / pathology
  • Male
  • Mutation* / genetics
  • Osmolar Concentration
  • Phenotype
  • Retrospective Studies
  • Semen / metabolism
  • Semen / physiology
  • Seminal Vesicles / pathology
  • Sperm Count
  • Testis / pathology
  • Ultrasonography
  • Vas Deferens / abnormalities*
  • Vas Deferens / diagnostic imaging
  • Vas Deferens / pathology

Substances

  • CFTR protein, human
  • Cystic Fibrosis Transmembrane Conductance Regulator
  • Fructose