Milder childhood form of very long-chain acyl-CoA dehydrogenase deficiency in a 6-year-old Japanese boy

Eur J Pediatr. 2000 Dec;159(12):908-11. doi: 10.1007/pl00008368.

Abstract

We investigated the clinical and biochemical characteristics of a 6-year-old Japanese boy with very-long-chain acyl-CoA dehydrogenase (VLCAD) deficiency. He had hypoketotic hypoglycaemia, exercise- and fasting-induced lethargy, hepatomegaly and cardiomegaly. Significant laboratory findings included elevated plasma levels of creatine phosphokinase and acyl-carnitine and a fatty liver at biopsy suggesting a diagnosis of VLCAD deficiency.

Conclusion: The diagnosis of very long-chain acyl-CoA dehydrogenase deficiency was supported by the results of acyl-CoA dehydrogenase activity for C8 and C16 fatty acids in skin fibroblasts from the patient. Treatment with medium chain triglycerides and L-carnitine in the diet improved his hepatomegaly and cardiomegaly.

Publication types

  • Case Reports

MeSH terms

  • Acyl-CoA Dehydrogenase, Long-Chain / deficiency*
  • Cardiomegaly / complications
  • Carnitine / therapeutic use
  • Child
  • Fatty Liver / complications
  • Fatty Liver / diagnosis
  • Heart Failure / complications
  • Heart Failure / diagnosis
  • Hepatomegaly / complications
  • Humans
  • Male
  • Metabolism, Inborn Errors / complications
  • Metabolism, Inborn Errors / diagnosis*
  • Metabolism, Inborn Errors / enzymology
  • Metabolism, Inborn Errors / therapy

Substances

  • Acyl-CoA Dehydrogenase, Long-Chain
  • Carnitine