HLA-G expression by tumors

Am J Reprod Immunol. 2001 Feb;45(2):103-7. doi: 10.1111/j.8755-8920.2001.450207.x.

Abstract

Problem: It has been proposed that the expression of the non-classical MHC class I antigen, HLA-G, by trophoblast is one mechanism by which the placenta evades attack by maternal uterine NK cells. A similar mechanism is thought to be operative in the escape from immunosurveillance by tumor cells. However, data on the expression of HLA-G by tumor cells are highly conflicting.

Method of study: In the present study, we have examined tissue sections from a wide variety of tumors by immunohistology and also several cell lines by flow cytometry and RT-PCR. Whilst very faint bands were detected in three cell lines (hepG2, Mead, CaSki) by RT-PCR, no tumors or cell lines were observed to express HLA-G protein. Furthermore, we found that tumor deposits are not usually infiltrated by NK cells.

Conclusion: Our observations, therefore, do not support the proposal that tumor cells express HLA-G in order to evade host NK cell cytolysis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • HLA Antigens / analysis*
  • HLA Antigens / genetics
  • HLA-G Antigens
  • Histocompatibility Antigens Class I / analysis*
  • Histocompatibility Antigens Class I / genetics
  • Humans
  • Immunohistochemistry
  • Leukocytes / physiology
  • Neoplasms / chemistry*
  • Neoplasms / pathology
  • RNA, Messenger / analysis
  • Tumor Cells, Cultured

Substances

  • HLA Antigens
  • HLA-G Antigens
  • Histocompatibility Antigens Class I
  • RNA, Messenger