Specific down-regulation of HER-2/neu mediated by a chimeric U6 hammerhead ribozyme results in growth inhibition of human ovarian carcinoma

Mol Ther. 2001 Feb;3(2):169-77. doi: 10.1006/mthe.2000.0241.

Abstract

The U6 expression system was explored for efficient expression of a ribozyme against the human proto-oncogene c-neu. A hammerhead ribozyme (neuRz) and the control mutant ribozyme (MRz) were targeted to cleave c-neu mRNA at the tyrosine kinase domain. In vitro cleavage showed that neuRz was very active while MRz was not. Near-maximal target cleavage observed at a low ribozyme:target ratio (0.1) suggests that neuRz has good activity and turnover capability under physiological conditions, i.e., <5 mM MgCl(2) and 37 degrees C. Chimeric U6 ribozyme was expressed at about 5 x 10(6) copies/cell at 48 h in the ovarian carcinoma cell line SKOV-3.ip1. Partial down-regulation of c-neu mRNA and protein was observed in a dose-dependent manner in cells transiently transfected with U6neuRz- but not with MRz-containing plasmid. Sorted transient transfectants demonstrated dramatic growth inhibition with the neuRz-expressing cells. Our results demonstrate that the U6 expression system is very efficient and suitable for the expression of a hammerhead ribozyme. Moreover, nonviral delivery of the neuRz-expressing plasmid resulted in specific down-regulation of c-neu and, subsequently, growth inhibition of ovarian cancer cells overexpressing c-neu.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Blotting, Northern
  • Cell Division
  • Cell Separation
  • Dose-Response Relationship, Drug
  • Down-Regulation*
  • Female
  • Flow Cytometry
  • Genetic Therapy / methods
  • Humans
  • Ovarian Neoplasms / metabolism*
  • Plasmids / metabolism
  • Promoter Regions, Genetic
  • Protein Structure, Tertiary
  • Protein-Tyrosine Kinases / chemistry
  • Proto-Oncogene Mas
  • RNA, Messenger / metabolism
  • Receptor, ErbB-2 / metabolism*
  • Ribonucleoprotein, U4-U6 Small Nuclear / metabolism*
  • Time Factors
  • Transfection
  • Tumor Cells, Cultured

Substances

  • MAS1 protein, human
  • Proto-Oncogene Mas
  • RNA, Messenger
  • Ribonucleoprotein, U4-U6 Small Nuclear
  • Protein-Tyrosine Kinases
  • Receptor, ErbB-2