Interleukin-15 expression in cutaneous T-cell lymphoma (mycosis fungoides and Sézary syndrome)

Br J Dermatol. 2001 May;144(5):1016-23. doi: 10.1046/j.1365-2133.2001.04192.x.

Abstract

Background: Cytokines are of potential importance in the pathogenesis of cutaneous T-cell mediated disorders, including cutaneous T-cell lymphoma (CTCL).

Objectives: To compare interleukin (IL)-15 expression in certain inflammatory cutaneous diseases, with that in CTCL (mycosis fungoides and Sézary syndrome).

Methods: IL-15 mRNA and protein expression were examined by in situ hybridization and immunohistochemistry, respectively, on formalin-fixed, paraffin-embedded biopsies of normal human skin, atopic dermatitis, psoriasis, parapsoriasis and CTCL.

Results: Despite similar expression of IL-15 mRNA, we found differences in IL-15 protein expression between normal human skin, atopic dermatitis and psoriasis on the one hand, and parapsoriasis and CTCL on the other. IL-15 protein expression was not detected in normal human skin, atopic dermatitis or psoriasis, but was detected, mainly at low levels but in a few patients at higher levels, in epidermal keratinocytes in parapsoriasis, mycosis fungoides and Sézary syndrome.

Conclusions: Induction of keratinocyte IL-15 expression appears to be a feature of CTCL. The factors stimulating such an expression remain unknown.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / therapeutic use
  • Dermatitis, Atopic / immunology
  • Dermatitis, Atopic / therapy
  • Gene Expression
  • Humans
  • Immunoenzyme Techniques
  • In Situ Hybridization
  • Interferon-alpha / therapeutic use
  • Interleukin-15 / genetics
  • Interleukin-15 / metabolism*
  • Mycosis Fungoides / immunology*
  • Mycosis Fungoides / therapy
  • Parapsoriasis / immunology
  • RNA, Messenger / genetics
  • Sezary Syndrome / immunology*
  • Sezary Syndrome / therapy
  • Skin / immunology
  • Skin Neoplasms / immunology*
  • Skin Neoplasms / therapy

Substances

  • Antineoplastic Agents
  • Interferon-alpha
  • Interleukin-15
  • RNA, Messenger