Cytokine production by mouse myeloid dendritic cells in relation to differentiation and terminal maturation induced by lipopolysaccharide or CD40 ligation

Blood. 2001 Sep 1;98(5):1512-23. doi: 10.1182/blood.v98.5.1512.

Abstract

Although it is known that dendritic cells (DCs) produce cytokines, there is little information about how cytokine synthesis is regulated during DC development. A range of cytokine mRNA/proteins was analyzed in immature (CD86-) or mature (CD86+) murine bone marrow (BM)- derived DCs. Highly purified, flow-sorted, immature DCs exhibited higher amounts of interleukin-1alpha (IL-1alpha), IL-1beta, tumor necrosis factor-alpha (TNF-alpha), transforming growth factor beta1 (TGF-beta1), and macrophage migration inhibitory factor (MIF) mRNA/protein than mature DCs. After differentiation, DC up-regulated the levels of IL-6 and IL-15 mRNA/protein and synthesized de novo mRNA/protein for IL-12p35, IL-12p40, and IL-18. Although immature BM-derived DCs did not stimulate naive allogeneic T cells, mature DCs elicited a mixed population of T helper (Th) 1 (mainly) and Th2 cells in 3d-mixed leukocyte reactions. CD86+ BM DCs switched to different cytokine patterns according to whether they were terminally differentiated by lipopolysaccharide (LPS) or CD40 ligation. Although both stimuli increased IL-6, IL-12p40, IL-15, and TNF-alpha mRNA/protein levels, only LPS up-regulated transcription of IL-1alpha, IL-1beta, IL-12p35, and MIF genes. Although LPS and CD40 cross-linking increased the T-cell allostimulatory function of BM DCs, only LPS stimulation shifted the balance of naive Th differentiation to Th1 cells, a mechanism dependent on the up-regulation of IL-12p35 and not of IL-23. These results demonstrate that, depending on the stimuli used to terminally mature BM DCs, DCs synthesize a different pattern of cytokines and exhibit distinct Th cell-driving potential.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antibodies, Monoclonal / pharmacology
  • Antigens, CD / biosynthesis
  • B7-2 Antigen
  • Bone Marrow Cells / drug effects
  • Bone Marrow Cells / metabolism
  • CD40 Antigens / immunology
  • CD40 Antigens / physiology*
  • Cell Differentiation / drug effects
  • Cells, Cultured / metabolism
  • Cytokines / biosynthesis*
  • Dendritic Cells / drug effects
  • Dendritic Cells / metabolism*
  • Endocytosis
  • Gene Expression Profiling
  • Gene Expression Regulation / drug effects*
  • Immunophenotyping
  • Interleukin-12 / physiology
  • Interleukin-23
  • Interleukin-23 Subunit p19
  • Interleukins / biosynthesis
  • Interleukins / genetics
  • Interleukins / physiology
  • Lipopolysaccharides / pharmacology*
  • Lymphocyte Culture Test, Mixed
  • Macrophage Migration-Inhibitory Factors / biosynthesis
  • Macrophage Migration-Inhibitory Factors / genetics
  • Membrane Glycoproteins / biosynthesis
  • Mice
  • Mice, Inbred C3H
  • Mice, Inbred C57BL
  • RNA, Messenger / biosynthesis
  • Specific Pathogen-Free Organisms
  • Th1 Cells / immunology
  • Th2 Cells / immunology
  • Transforming Growth Factor beta / biosynthesis
  • Transforming Growth Factor beta / genetics
  • Tumor Necrosis Factor-alpha / biosynthesis
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Antibodies, Monoclonal
  • Antigens, CD
  • B7-2 Antigen
  • CD40 Antigens
  • Cd86 protein, mouse
  • Cytokines
  • Il23a protein, mouse
  • Interleukin-23
  • Interleukin-23 Subunit p19
  • Interleukins
  • Lipopolysaccharides
  • Macrophage Migration-Inhibitory Factors
  • Membrane Glycoproteins
  • RNA, Messenger
  • Transforming Growth Factor beta
  • Tumor Necrosis Factor-alpha
  • Interleukin-12