Gene polymorphism in Netherton and common atopic disease

Nat Genet. 2001 Oct;29(2):175-8. doi: 10.1038/ng728.

Abstract

Atopic dermatitis (AD) and asthma are characterized by IgE-mediated atopic (allergic) responses to common proteins (allergens), many of which are proteinases. Loci influencing atopy have been localized to a number of chromosomal regions, including the chromosome 5q31 cytokine cluster. Netherton disease is a rare recessive skin disorder in which atopy is a universal accompaniment. The gene underlying Netherton disease (SPINK5) encodes a 15-domain serine proteinase inhibitor (LEKTI) which is expressed in epithelial and mucosal surfaces and in the thymus. We have identified six coding polymorphisms in SPINK5 (Table 1) and found that a Glu420-->Lys variant shows significant association with atopy and AD in two independent panels of families. Our results implicate a previously unrecognized pathway for the development of common allergic illnesses.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Asthma / genetics*
  • Base Sequence
  • Carrier Proteins*
  • DNA Primers
  • Dermatitis, Atopic / genetics*
  • Humans
  • Molecular Sequence Data
  • Polymorphism, Single Nucleotide*
  • Proteinase Inhibitory Proteins, Secretory
  • Sequence Homology, Amino Acid
  • Serine Peptidase Inhibitor Kazal-Type 5
  • Serine Proteinase Inhibitors / genetics*

Substances

  • Carrier Proteins
  • DNA Primers
  • Proteinase Inhibitory Proteins, Secretory
  • SPINK5 protein, human
  • Serine Peptidase Inhibitor Kazal-Type 5
  • Serine Proteinase Inhibitors