Postischemic (6-Hour) treatment with recombinant human tissue plasminogen activator and proteasome inhibitor PS-519 reduces infarction in a rat model of embolic focal cerebral ischemia

Stroke. 2001 Dec 1;32(12):2926-31. doi: 10.1161/hs1201.100207.

Abstract

Background and purpose: The proteasome inhibitor PS-519 blocks activation of nuclear factor-kappaB, a major mediator of inflammation. We tested the hypothesis that combination treatment of recombinant human tissue plasminogen activator (rhtPA) and PS-519 extends the therapeutic window for treatment of stroke with rhtPA without increasing incidence of hemorrhagic transformation.

Methods: The middle cerebral artery (MCA) of male Wistar rats (n=56) was occluded by an embolus. After embolization, animals were randomly divided into the following groups: PS-519 treatment groups: PS-519 was given at 2, 4, or 6 hours after MCA occlusion; rhtPA treatment groups: rhtPA was given at 2 or 4 hours after MCA occlusion; combination treatment groups: PS-519 and rhtPA were given at 2, 4, or 6 hours after MCA occlusion; control group: the same volume of saline was given at 2 hours after MCA occlusion.

Results: Administration of PS-519 alone at 2 or 4 hours, but not 6 hours, significantly (P<0.05) reduced infarct volume and improved neurological recovery compared with the control group. Administration of rhtPA alone at 2 hours, but not 4 hours, significantly (P<0.05) reduced infarct volume and improved neurological recovery compared with the control group. Furthermore, combination treatment with rhtPA and PS-519 even at 6 hours significantly (P<0.05) reduced infarct volume, improved neurological recovery, and did not increase the incidence of hemorrhagic transformation compared with the control group or the group treated with PS-519 alone.

Conclusions: Our data suggest that combination treatment with PS-519 and rhtPA extends the neuroprotective effect to at least 6 hours after embolization.

MeSH terms

  • Acetylcysteine / administration & dosage*
  • Acetylcysteine / analogs & derivatives*
  • Animals
  • Behavior, Animal / drug effects
  • Blood Pressure / drug effects
  • Body Weight / drug effects
  • Brain Ischemia / complications
  • Brain Ischemia / drug therapy*
  • Brain Ischemia / metabolism
  • Brain Ischemia / pathology
  • Cell Count
  • Cerebral Hemorrhage / etiology
  • Cerebral Hemorrhage / pathology
  • Cerebral Hemorrhage / prevention & control
  • Cerebral Infarction / etiology
  • Cerebral Infarction / pathology
  • Cerebral Infarction / prevention & control*
  • Cysteine Endopeptidases
  • Cysteine Proteinase Inhibitors / administration & dosage
  • Disease Models, Animal
  • Drug Administration Schedule
  • Drug Therapy, Combination
  • Fibrinolytic Agents / administration & dosage
  • Fibrinolytic Agents / adverse effects
  • Humans
  • Intracranial Embolism / complications
  • Intracranial Embolism / drug therapy*
  • Intracranial Embolism / pathology
  • Male
  • Multienzyme Complexes / antagonists & inhibitors
  • Neurologic Examination
  • Peroxidase / metabolism
  • Proteasome Endopeptidase Complex
  • Rats
  • Rats, Wistar
  • Recombinant Proteins / administration & dosage
  • Recombinant Proteins / adverse effects
  • Time Factors
  • Tissue Plasminogen Activator / administration & dosage*
  • Tissue Plasminogen Activator / adverse effects

Substances

  • Cysteine Proteinase Inhibitors
  • Fibrinolytic Agents
  • Multienzyme Complexes
  • PS519
  • Recombinant Proteins
  • Peroxidase
  • Tissue Plasminogen Activator
  • Cysteine Endopeptidases
  • Proteasome Endopeptidase Complex
  • Acetylcysteine