Multidrug resistance mechanisms in chronic lymphocytic leukaemia

Br J Haematol. 2002 Mar;116(4):774-80. doi: 10.1046/j.0007-1048.2002.03344.x.

Abstract

We evaluated the presence of P-glycoprotein (P-gp)-170, multidrug resistance protein (MRP), lung resistance protein (LRP)-56 and Bcl-2 in CD19-positive cells from 100 cases of chronic lymphocytic leukaemia (CLL). P-gp-170 was found in 73% of the CLL cases with no significant difference regarding stage or previous treatment. LRP-56 protein was homogeneously distributed with no differences for stage or treatment. MRP protein was detected at a low level of expression in 49.4% of CLL patients with no differences for stage or treatment. Bcl-2 protein was expressed at a high level in all CLL patients and higher levels were found in the advanced stage. This leads us to conclude that P-gp, MRP, LRP-56 and Bcl-2 are frequently expressed in CLL. P-gp, MRP and LRP are not correlated to stage or previous treatment. Bcl-2 is higher in advanced-stage patients. The clinical and biological significance of these zMDR mechanisms in CLL remains to be fully explained.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • ATP Binding Cassette Transporter, Subfamily B, Member 1 / analysis
  • Antigens, CD19
  • Drug Resistance, Multiple*
  • Humans
  • Leukemia, Lymphocytic, Chronic, B-Cell / drug therapy
  • Leukemia, Lymphocytic, Chronic, B-Cell / metabolism*
  • Lymphocytes / chemistry
  • Neoplasm Proteins / analysis
  • Proto-Oncogene Proteins c-bcl-2 / analysis
  • Vault Ribonucleoprotein Particles

Substances

  • ATP Binding Cassette Transporter, Subfamily B, Member 1
  • Antigens, CD19
  • Neoplasm Proteins
  • Proto-Oncogene Proteins c-bcl-2
  • Vault Ribonucleoprotein Particles
  • major vault protein