Enhanced learning and memory and altered GABAergic synaptic transmission in mice lacking the alpha 5 subunit of the GABAA receptor

J Neurosci. 2002 Jul 1;22(13):5572-80. doi: 10.1523/JNEUROSCI.22-13-05572.2002.

Abstract

The alpha5 subunit of the GABA(A) receptor is localized mainly to the hippocampus of the mammalian brain. The significance of this rather distinct localization and the function of alpha5-containing GABA(A) receptors has been explored by targeted disruption of the alpha5 gene in mice. The alpha5 -/- mice showed a significantly improved performance in a water maze model of spatial learning, whereas the performance in non-hippocampal-dependent learning and in anxiety tasks were unaltered in comparison with wild-type controls. In the CA1 region of hippocampal brain slices from alpha5 -/- mice, the amplitude of the IPSCs was decreased, and paired-pulse facilitation of field EPSP (fEPSP) amplitudes was enhanced. These data suggest that alpha5-containing GABA(A) receptors play a key role in cognitive processes by controlling a component of synaptic transmission in the CA1 region of the hippocampus.

MeSH terms

  • Animals
  • Avoidance Learning
  • Behavior, Animal
  • Electric Conductivity
  • Excitatory Postsynaptic Potentials
  • Female
  • Hippocampus / physiology*
  • Kinetics
  • Learning*
  • Long-Term Potentiation
  • Male
  • Maze Learning
  • Memory*
  • Mice
  • Mice, Knockout
  • Protein Subunits
  • Receptors, GABA-A / genetics
  • Receptors, GABA-A / physiology*
  • Synaptic Transmission*
  • gamma-Aminobutyric Acid / metabolism

Substances

  • Protein Subunits
  • Receptors, GABA-A
  • gamma-Aminobutyric Acid