657del5 mutation in the NBS1 gene is associated with Nijmegen breakage syndrome in a Turkish family

Clin Genet. 2002 Jul;62(1):84-8. doi: 10.1034/j.1399-0004.2002.620112.x.

Abstract

We report on a consanguineous Turkish family whose first son died of anal atresia and whose second son presented with severe pre- and post-natal growth retardation as well as striking microcephaly, immunodeficiency, congenital heart disease, chromosomal instability and rhabdomyosarcoma in the anal region. The proband was found to carry the homozygous 657del5 mutation in the NBS1 gene, which is responsible for Nijmegen breakage syndrome (NBS) in most of the Slav populations. Our family, the first diagnosed with NBS in the Turkish population, represents one of the most severely affected examples of the syndrome, with profound pre- and post-natal growth retardation associated with structural abnormalities, and expands the clinical spectrum of this rare disorder.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Abnormalities, Multiple / genetics*
  • Adult
  • Cell Cycle Proteins / genetics*
  • Child, Preschool
  • Chromosome Disorders
  • Chromosomes, Human, Pair 14
  • Chromosomes, Human, Pair 15
  • Chromosomes, Human, Pair 7
  • Female
  • Humans
  • Infant, Newborn
  • Male
  • Nuclear Proteins / genetics*
  • Sequence Deletion*
  • Turkey

Substances

  • Cell Cycle Proteins
  • NBN protein, human
  • Nuclear Proteins