Association of an intronic polymorphism in the midkine (MK) gene with human sporadic colorectal cancer

Cancer Lett. 2002 Jun 28;180(2):159-63. doi: 10.1016/s0304-3835(02)00040-x.

Abstract

Midkine (MK) is a heparin-binding growth factor specified by a retinoic acid responsive gene. It plays important roles in development and carcinogenesis. The MK gene is located on chromosome 11q11.2 in humans. A heterozygous G to T transition at the 62nd base in intron 3 of this gene has been identified in sporadic colorectal and gastric cancers (Int. J. Mol. Med. 6 (2000) 281). To clarify whether this polymorphism is associated with a cancer risk, a case-control study was conducted. We examined 98 colorectal, 60 gastric, 59 esophagus, 32 lung and 37 breast cancer tissue specimens and their corresponding non-neoplastic tissues. Also, 86 unaffected control specimens were examined. The G/T genotype frequency in colorectal cancers was higher than that in normal samples (11.2 versus 2.3%; P=0.017). Therefore, this genotype could represent a risk factor for tumorigenesis in the colon and rectum of Japanese.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Carrier Proteins / genetics*
  • Colorectal Neoplasms / etiology
  • Colorectal Neoplasms / genetics*
  • Cytokines*
  • Female
  • Humans
  • Introns*
  • Male
  • Midkine
  • Polymorphism, Genetic
  • Risk Factors

Substances

  • Carrier Proteins
  • Cytokines
  • Midkine