Identification of a conserved GATA3 response element upstream proximal from the interleukin-13 gene locus

J Biol Chem. 2002 Nov 1;277(44):42399-408. doi: 10.1074/jbc.M205876200. Epub 2002 Aug 29.

Abstract

Differentiation of naive CD4 T cells into type 2 helper (Th2) cells is accompanied by chromatin remodeling of Th2 cytokine gene loci. Hyperacetylation of histone H3 on nucleosomes associated with the interleukin (IL)-4, IL-13 and IL-5 genes was observed in developing Th2 cells but not in Th1 cells. Histone hyperacetylation on IL-5 gene-associated nucleosomes was Th2-specific but occurred with delayed kinetics, and hyperacetylation on RAD50 gene-associated nucleosomes was T cell antigen receptor stimulation-dependent but not Th2-specific. The induction of the Th2-specific histone hyperacetylation was STAT6- and GATA3-dependent, and interestingly, it was accompanied by the expression of intergenic transcripts within the IL-13 and IL-4 gene loci. A conserved GATA3 response element (CGRE) containing four GATA consensus sequences was identified 1.6 kbp upstream from the IL-13 gene, corresponding with the 5'-border of the Th2-specific histone hyperacetylation region. The CGRE was shown to bind to GATA3, histone acetyltransferase complexes including CBP/p300, and RNA polymerase II. Also, the CGRE showed a significant enhancing effect on the Th2 cytokine gene promoters. Thus, the CGRE may play a crucial role for GATA3-mediated targeting and downstream spreading of core histone hyperacetylation within the IL-13 and IL-4 gene loci.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylation
  • Animals
  • Cells, Cultured
  • Chromosome Mapping*
  • DNA-Binding Proteins / physiology*
  • Enhancer Elements, Genetic / physiology
  • Exons
  • GATA3 Transcription Factor
  • Histones / metabolism
  • Interleukin-13 / genetics*
  • Interleukin-4 / genetics
  • Mice
  • Mice, Inbred C57BL
  • Nucleosomes / metabolism
  • Response Elements / physiology*
  • STAT6 Transcription Factor
  • Th2 Cells / metabolism
  • Trans-Activators / physiology*
  • Transcription, Genetic

Substances

  • DNA-Binding Proteins
  • GATA3 Transcription Factor
  • Gata3 protein, mouse
  • Histones
  • Interleukin-13
  • Nucleosomes
  • STAT6 Transcription Factor
  • Stat6 protein, mouse
  • Trans-Activators
  • Interleukin-4