Adenovirus-mediated combined P16 gene and GM-CSF gene therapy for the treatment of established tumor and induction of antitumor immunity

Cancer Gene Ther. 2002 Oct;9(10):819-24. doi: 10.1038/sj.cgt.7700502.

Abstract

Antitumor effects of combined transfer of P16 and cytokine genes were investigated in this study. The adenovirus harboring the P16 gene (AdP16) and murine granulocyte-macrophage colony-stimulating factor gene (AdGM-CSF) were utilized for the treatment of established tumors. The mice were inoculated subcutaneously with Renca cells and, 6 days later, received an intratumoral injection of AdP16 in the presence or absence of AdGM-CSF. The results demonstrated that tumor-bearing mice treated with AdP16 in combination with AdGM-CSF showed more potent inhibition of tumor growth and survived much longer than did mice treated with AdP16, AdGM-CSF, adenovirus expressing beta-galactosidase, or phosphate-buffered saline alone (P<.01). The tumor mass showed obvious necrosis and inflammatory cell infiltration, and more CD(4)(+) and CD(8)(+) T cells infiltrating the tumor after combined therapy. After combined therapy, the expression of MHC-1 (H-2K(d)) and Fas molecules on freshly isolated tumor cells increased greatly. The activity of specific cytotoxic T lymphocytes was also found to be induced more significantly after the combined therapy (P<.01). Our results demonstrated that combined therapy with P16 and GM-CSF genes can inhibit the growth of established tumors in mice significantly and induce antitumor immunity of the host efficiently.

Publication types

  • Comparative Study

MeSH terms

  • Adenoviridae / genetics*
  • Animals
  • Carcinoma, Renal Cell / immunology*
  • Carcinoma, Renal Cell / therapy
  • Combined Modality Therapy
  • DNA, Recombinant
  • Female
  • Flow Cytometry
  • Gene Transfer Techniques
  • Genes, p16*
  • Genetic Therapy / methods*
  • Genetic Vectors
  • Granulocyte-Macrophage Colony-Stimulating Factor / genetics*
  • H-2 Antigens / immunology
  • Immunity, Cellular
  • In Vitro Techniques
  • Kidney Neoplasms / immunology*
  • Kidney Neoplasms / therapy
  • Killer Cells, Natural / immunology
  • Mice
  • Mice, Inbred BALB C
  • T-Lymphocytes / immunology
  • T-Lymphocytes, Cytotoxic / immunology
  • Transfection
  • Tumor Cells, Cultured
  • fas Receptor / metabolism

Substances

  • DNA, Recombinant
  • H-2 Antigens
  • H-2K(K) antigen
  • fas Receptor
  • Granulocyte-Macrophage Colony-Stimulating Factor