Abstract
Dendritic cells (DC) control immunity and tolerance. Hence, we surmised that systemic lupus erythematosus (SLE), a systemic autoimmune disease with autoreactive T and B cells, might be due to DC alterations. Based on our findings, we are proposing a model of SLE where autoimmune responses are driven by unabated activation of myeloid DC through IFN-alpha produced by plasmacytoid DC. Thus, interplay between DC subsets might represent a key component of SLE pathogenesis.
MeSH terms
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Autoantibodies / biosynthesis
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Autoimmune Diseases / immunology*
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B-Lymphocyte Subsets / immunology
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CD11c Antigen / analysis
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Cytokines / physiology
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Dendritic Cells / classification
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Dendritic Cells / immunology*
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Dendritic Cells / metabolism
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Homeostasis
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Humans
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Interferon-alpha / physiology
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Lupus Erythematosus, Systemic / immunology*
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Models, Immunological
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Myeloid Cells / immunology
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T-Lymphocyte Subsets / immunology
Substances
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Autoantibodies
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CD11c Antigen
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Cytokines
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Interferon-alpha