A functional thrombin receptor (PAR1) is expressed on bone-derived prostate cancer cell lines

Urology. 2002 Nov;60(5):760-5. doi: 10.1016/s0090-4295(02)01969-6.

Abstract

Objectives: To identify genes important in prostate cancer metastatic to bone. Bone-specific metastasis is a common feature of prostate cancer and a significant cause of morbidity.

Methods: To identify factors involved in organ-specific metastasis, we used cDNA microarray analysis to compare a bone-derived cell line, VCaP, with a soft tissue-derived cell line, DuCaP. Both cell lines were derived from the same patient and spontaneously passaged.

Results: Forty-five genes were differentially expressed, and only seven of these also had increased expression in VCaP compared with normal prostatic tissue. Of these, protease-activated receptor 1 (PAR1) was verified as having increased expression by reverse transcriptase-polymerase chain reaction and Northern blot analysis, as well as by immunohistochemistry. PAR1 expression in a panel of prostate cancer cell lines demonstrated increased expression in those cell lines derived from bone metastases. Alpha-thrombin stimulation of the VCaP cells produced a dose-dependent mobilization of intracellular calcium compared with DuCaP, suggesting that PAR1 expressed on the VCaP prostate cancer cell line is functional.

Conclusions: These data indicate that a functional PAR1 is expressed on prostate cancer cell lines. The prostate cancer cell lines expressing PAR1 appear to have an association with increased bone metastases.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Bone Neoplasms / genetics*
  • Bone Neoplasms / secondary*
  • Calcium / metabolism
  • Gene Expression Regulation, Neoplastic
  • Hemostatics / pharmacology
  • Humans
  • Male
  • Neoplasm Proteins / genetics*
  • Neoplasm Proteins / metabolism
  • Oligonucleotide Array Sequence Analysis
  • Organ Specificity
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / pathology
  • Receptor, PAR-1
  • Receptors, Thrombin / genetics*
  • Receptors, Thrombin / metabolism
  • Thrombin / pharmacology
  • Tumor Cells, Cultured

Substances

  • Hemostatics
  • Neoplasm Proteins
  • Receptor, PAR-1
  • Receptors, Thrombin
  • Thrombin
  • Calcium