Abstract
Recently, it was shown that t(10;16)(q22;p13) fuses the MORF and CREBBP genes in a case of childhood acute myeloid leukemia (AML) M5a, with a complex karyotype containing other rearrangements. Here, we report a new case with the MORF-CREBBP fusion in an 84-year-old patient diagnosed with AML M5b, in which the t(10;16)(q22;p13) was the only cytogenetic aberration. This supports that this is a recurrent pathogenic translocation in AML.
Copyright 2003 Wiley-Liss, Inc.
MeSH terms
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Acetyltransferases / genetics*
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Aged
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Aged, 80 and over
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CREB-Binding Protein
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Chromosomes, Human, Pair 10 / genetics*
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Chromosomes, Human, Pair 16 / genetics*
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Cytogenetic Analysis / methods
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Fatal Outcome
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Histone Acetyltransferases
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Humans
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Leukemia, Monocytic, Acute / diagnosis
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Leukemia, Monocytic, Acute / genetics*
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Male
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Neoplasm Recurrence, Local / genetics
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Nuclear Proteins / genetics*
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Oncogene Proteins, Fusion / genetics*
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Saccharomyces cerevisiae Proteins / genetics*
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Trans-Activators / genetics*
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Translocation, Genetic / genetics
Substances
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Nuclear Proteins
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Oncogene Proteins, Fusion
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Saccharomyces cerevisiae Proteins
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Trans-Activators
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Acetyltransferases
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CREB-Binding Protein
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CREBBP protein, human
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Histone Acetyltransferases