[Inhibitory effect of artemin on endotoxin-induced nitric oxide synthesis]

Zhongguo Zhong Yao Za Zhi. 2001 Nov;26(11):770-3.
[Article in Chinese]

Abstract

Objective: To investigate the effect of Artemin on LPS-induced nitric oxide synthesis in macrophages.

Method: 1. Nitrite oxide (NO) production of RAW 264.7 cells was induced by LPS or LPS in combination with interferon-gamma (IFN gamma) in the presence or absence of Artemin. The amount of NO in the supernatant of RAW 264.7 cells was detected with Griess reagent. 2. Balb/c mice were injected with Artemin (i.m.) 50 mg.kg-1.d-1 x 3 d, and intraperitoneal macrophages were collected to detect the LPS-induced NO production.

Result: LPS 1.0, 0.2 microgram.ml-1 or IFN-gamma 100u + LPS 1.0, 0.2, 0.04 microgram.ml-1 could induced a large amount of NO synthesis of RAW 264.7 cells. Artemin showed a significant inhibitory effect on LPS or IFN-gamma + LPS-induced NO production in a dose dependent manner. After treatment with Artemin, the response of Balb/c mice to LPS was reduced, which was showed by a decrease in NO production of intraperitoneal macrophages induced by LPS.

Conclusion: Artemin could reduce LPS-induced production of inflammatory factors resulting in the inhibition of inflammatory effects.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line
  • Dose-Response Relationship, Drug
  • Interferon-gamma / antagonists & inhibitors
  • Lipopolysaccharides / antagonists & inhibitors
  • Macrophages / cytology
  • Macrophages / metabolism
  • Macrophages, Peritoneal / metabolism*
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Nerve Tissue Proteins / pharmacology*
  • Nitric Oxide / biosynthesis*

Substances

  • Artn protein, mouse
  • Lipopolysaccharides
  • Nerve Tissue Proteins
  • Nitric Oxide
  • Interferon-gamma