Long-cycle structured intermittent versus continuous highly active antiretroviral therapy for the treatment of chronic infection with human immunodeficiency virus: effects on drug toxicity and on immunologic and virologic parameters

J Infect Dis. 2003 Aug 1;188(3):388-96. doi: 10.1086/376535. Epub 2003 Jul 10.

Abstract

We evaluated the effect of long-cycle structured intermittent therapy (SIT; 4 weeks without highly active antiretroviral therapy [HAART] followed by 8 weeks with HAART) versus continuous HAART. The study was prematurely terminated to new enrollment because of the emergence of genetic mutations associated with resistance to antiretroviral drugs in 5 patients. After 48 weeks, there was no significant difference between groups in lipid, hepatic transaminase, and C-reactive protein levels in 41 patients. Although there were no differences in CD4(+) or CD8(+) T cell counts or the percentage of cells that were CD4(+)CD25(+), CD8(+)CD25(+), or CD4(+)DR(+), patients who received SIT had a significantly higher percentage of CD8(+)CD38(+) and CD8(+)DR(+) cells. There was no clear autoimmunization effect by immunologic or virologic parameters. There was no benefit to long-cycle SIT versus continuous HAART with regard to certain toxicity, immunologic, or virologic parameters.

Publication types

  • Clinical Trial
  • Comparative Study
  • Randomized Controlled Trial

MeSH terms

  • Alanine Transaminase / blood
  • Aminopeptidases / blood
  • Anti-HIV Agents / administration & dosage
  • Anti-HIV Agents / therapeutic use*
  • Antiretroviral Therapy, Highly Active*
  • C-Reactive Protein / analysis
  • CD4-CD8 Ratio
  • Drug Administration Schedule
  • Drug Resistance, Viral
  • Follow-Up Studies
  • Glutamyl Aminopeptidase
  • HIV Infections / blood
  • HIV Infections / drug therapy*
  • HIV Infections / immunology
  • Humans
  • Lipids / blood
  • Lymphocyte Count
  • RNA, Viral / analysis
  • Receptors, Interleukin-2 / analysis
  • T-Lymphocytes / immunology
  • Treatment Outcome

Substances

  • Anti-HIV Agents
  • Lipids
  • RNA, Viral
  • Receptors, Interleukin-2
  • C-Reactive Protein
  • Alanine Transaminase
  • Aminopeptidases
  • Glutamyl Aminopeptidase