A promoter SNP (-1323T>C) in G-substrate gene (GSBS) correlates with hypercholesterolemia

J Hum Genet. 2003;48(9):447-450. doi: 10.1007/s10038-003-0055-x. Epub 2003 Sep 3.

Abstract

Factors predisposing to the phenotypic features of higher total cholesterol (TC) have not been clearly defined. Here we report an association between a promoter SNP (-1323T>C) in G-substrate gene (GSBS) and TC levels in 368 adult individuals from an east-central area of Japan. Age and gender-adjusted levels of LDL-cholesterol, TG, TC, and HDL-cholesterol were analyzed. When we separate the subjects into two genotypic groups regarding T allele, those who bear the T allele had significantly higher plasma TC levels than the others who lack the T allele (mean; 239.6 mg/dl vs. 210.6 mg/dl; p=0.003; Mann-Whitney test). Of the 341 individuals with the T allele, approximately 80% individuals presented with hypercholesterolemia, whereas only 44% were hypercholesterolemic among the 27 individuals without the T allele (p=0.0001). These results indicate a significant elevating effect of plasma TC levels by a SNP in the putative regulatory region of the G-substrate gene in our studied population. These data suggest that genetic variation at the G-substrate gene may be one of the determinants for plasma lipoprotein levels.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cholesterol / blood
  • Cholesterol, HDL / blood
  • Cholesterol, LDL / blood
  • Cohort Studies
  • DNA Mutational Analysis
  • Female
  • Genotype
  • Humans
  • Hypercholesterolemia / genetics*
  • Japan
  • Male
  • Middle Aged
  • Nerve Tissue Proteins / genetics*
  • Phenotype
  • Polymerase Chain Reaction
  • Polymorphism, Single Nucleotide / genetics*
  • Promoter Regions, Genetic / genetics*
  • Protein Kinases / genetics*
  • Triglycerides / blood

Substances

  • Cholesterol, HDL
  • Cholesterol, LDL
  • G-substrate
  • Nerve Tissue Proteins
  • Triglycerides
  • Cholesterol
  • Protein Kinases