The phosphorylation state of DARPP-32, a third messenger for dopamine, is regulated by in vivo pharmacological treatments

Neurochem Int. 1992 Mar:20 Suppl:165S-170S. doi: 10.1016/0197-0186(92)90232-g.

Abstract

It has been recently proposed that DARPP-32 participates, as third messenger, in the mediation of effects induced by dopamine at the cellular level. DARPP-32 is indeed localized almost exclusively on dopaminoceptive neurons bearing the D1 receptor subtype and it is phosphorylated by cAMP-dependent protein kinase. In its phospho-form, DARPP-32 acts as an inhibitor of protein phosphatase-1. In vivo pharmacological treatment with selective D1 agonists and antagonists induces changes in the phosphorylation state of DARPP-32 that can be correlated to changes in cAMP, mediated in turn by D1 and D2 receptors. These data demonstrate that the measurement of the phosphorylation state of DARPP-32 with the back-phosphorylation assay can represent a useful biochemical tool to gain further insight into the sequence of events elicited by specific dopaminergic drugs in vivo.

Publication types

  • Review

MeSH terms

  • Animals
  • Dopamine / metabolism*
  • Dopamine and cAMP-Regulated Phosphoprotein 32
  • Nerve Tissue Proteins / metabolism*
  • Nerve Tissue Proteins / physiology
  • Neurons / physiology
  • Phosphoproteins / metabolism
  • Phosphorylation
  • Receptors, Dopamine / classification
  • Receptors, Dopamine / metabolism
  • Tissue Distribution

Substances

  • Dopamine and cAMP-Regulated Phosphoprotein 32
  • Nerve Tissue Proteins
  • Phosphoproteins
  • Receptors, Dopamine
  • Dopamine