An insert mutation in the chromosome 20 amyloid precursor gene in a Gerstmann-Sträussler-Scheinker family

J Neurol Sci. 1992 Sep;111(2):189-94. doi: 10.1016/0022-510x(92)90067-u.

Abstract

We report the finding of an insert mutation in the chromosome 20 amyloid precursor gene in a family with neuropathologically-verified, experimentally-transmitted Gerstmann-Sträussler-Scheinker syndrome (GSS). The insert consisted of 8 extra copies of a repeating octapeptide coding sequence in the region between codons 51 and 91; it was identified in the proband and a presently unaffected at-risk niece by full sequencing of the open reading frame, and was visualized electrophoretically in the proband and 6 of 12 at-risk relatives. Although affected members in this French-Breton family have shown a variety of clinical profiles, including durations of illness that ranged from 3 months to 13 years, all autopsied cases (including the patient with the shortest illness) have had the distinctive multicentric amyloid plaques that define GSS as a nosologic entity.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Amino Acid Sequence
  • Amyloid beta-Protein Precursor / genetics*
  • Animals
  • Base Sequence
  • Brain Tissue Transplantation / adverse effects
  • Chromosomes, Human, Pair 20*
  • Codon
  • Female
  • Gerstmann-Straussler-Scheinker Disease / genetics*
  • Gerstmann-Straussler-Scheinker Disease / transmission
  • Haplorhini
  • Humans
  • Male
  • Middle Aged
  • Molecular Sequence Data
  • Mutation
  • Pan troglodytes
  • Pedigree
  • Transplantation, Heterologous

Substances

  • Amyloid beta-Protein Precursor
  • Codon