Drug-selected resistance mutations and non-B subtypes in antiretroviral-naive adults with established human immunodeficiency virus infection

J Infect Dis. 2003 Oct 1;188(7):986-91. doi: 10.1086/378280. Epub 2003 Sep 10.

Abstract

The prevalence of human immunodeficiency virus (HIV) type 1 antiretroviral resistance is expected to be higher in recently infected antiretroviral-naive individuals than in those who have been infected longer. Antiretroviral-naive HIV-1-infected adults who presented to an outpatient clinic in an urban hospital in Boston for initial evaluation in 1999 were screened for drug-selected resistance mutations and phylogenetic subtype. Drug-selected mutations were identified in 16 (18%) of 88 subjects. Twelve (14%) included mutations associated with nucleoside reverse-transcriptase inhibitors, 4 (5%) included mutations associated with nonnucleoside reverse-transcriptase inhibitors, and 3 (3%) included mutations associated with protease inhibitors. Two (2%) had resistance mutations associated with multiple classes of drugs. Nine (10%) subjects had infection with non-B subtype HIV-1 and did not have drug-selected mutations. Serological results indicated infection for >/=6 months. Drug-selected mutations or non-B subtypes were detected in a substantial portion of antiretroviral-naive adults who had been infected for at least 6 months.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Anti-HIV Agents / adverse effects
  • Anti-HIV Agents / therapeutic use
  • Base Sequence
  • Boston
  • Drug Resistance, Viral / genetics*
  • HIV Infections / drug therapy
  • HIV Infections / virology*
  • HIV Protease / chemistry
  • HIV Protease / genetics
  • HIV Reverse Transcriptase / chemistry
  • HIV Reverse Transcriptase / genetics
  • HIV-1 / classification
  • HIV-1 / genetics*
  • Humans
  • Molecular Sequence Data
  • Phylogeny
  • Point Mutation / genetics
  • RNA, Viral / blood
  • Reverse Transcriptase Polymerase Chain Reaction
  • Selection, Genetic
  • Sequence Analysis, DNA
  • Viral Load

Substances

  • Anti-HIV Agents
  • RNA, Viral
  • HIV Reverse Transcriptase
  • HIV Protease