Hypercholesterolemia associated with splice-junction variation of inter-alpha-trypsin inhibitor heavy chain 4 (ITIH4) gene

J Hum Genet. 2004;49(1):24-28. doi: 10.1007/s10038-003-0101-8. Epub 2003 Dec 6.

Abstract

Factors predisposing to the phenotypic features of higher total cholesterol (T-Cho) have not been clearly defined. Here we report an association between a C/T single nucleotide polymorphism at IVS17+8 in the inter-alpha-trypsin inhibitor heavy chain 4 gene (ITIH4) and plasma total cholesterol levels in 351 adult individuals from an east-central area of Japan. Age and gender-adjusted levels of plasma T-Cho, LDL-cholesterol, triglyceride, and HDL-cholesterol were analyzed. When we separate the subjects into two genotypic groups regarding this single nucleotide polymorphism (SNP), those who lack the T-allele had significantly higher plasma T-Cho levels than the others who bear T-allele (mean 252.3 mg/dl versus 241.7 mg/dl; p=0.009). Of the 309 individuals without the T-allele, approximately 90% presented with hypercholesterolemia, whereas only 10% were hypercholesterolemic among 42 individuals with the T-allele (p <0.0001). These data suggest that genetic variation at ITIH4 locus is one of the likely candidate determinants for plasma cholesterol metabolisms.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Alpha-Globulins / genetics*
  • Analysis of Variance
  • Cholesterol / blood
  • Chromosomes, Human, Pair 3 / genetics
  • Cohort Studies
  • Female
  • Genetic Variation*
  • Humans
  • Hypercholesterolemia / blood
  • Hypercholesterolemia / genetics*
  • Japan
  • Male
  • Middle Aged
  • Polymorphism, Single Nucleotide / genetics*
  • Serine Proteinase Inhibitors / genetics*

Substances

  • Alpha-Globulins
  • Serine Proteinase Inhibitors
  • inter-alpha-inhibitor
  • Cholesterol