Nicotine enhances contextual fear conditioning and ameliorates ethanol-induced deficits in contextual fear conditioning

Behav Neurosci. 2003 Dec;117(6):1276-82. doi: 10.1037/0735-7044.117.6.1276.

Abstract

Nicotine and ethanol are 2 commonly used and abused drugs that have divergent effects on learning. The present study examined the effects of acute nicotine (0.25 mg/kg), ethanol (1.0 g/kg), and ethanol-nicotine coadministration on fear conditioning in C57BL/6 mice. Mice were assessed for contextual and cued fear conditioning at 1 day and 1 week posttraining. Ethanol disrupted acquisition but not consolidation of contextual fear conditioning; nicotine enhanced contextual fear conditioning and ameliorated ethanol-associated deficits in contextual fear conditioning. Mecamylamine antagonized this effect. Fear conditioning was reassessed 1 week after initial testing with no drug administered. At the 1-week retest, mice previously treated with nicotine continued to show enhanced contextual fear, and mice previously treated with ethanol continued to show contextual fear deficits. Thus, nicotine both produces a long-lasting enhancement of contextual fear conditioning and protects against ethanol-associated deficits.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Central Nervous System Depressants / pharmacology
  • Conditioning, Classical / drug effects*
  • Drug Interactions
  • Ethanol / pharmacology*
  • Fear / drug effects*
  • Male
  • Mecamylamine / pharmacology
  • Mental Recall / drug effects
  • Mice
  • Mice, Inbred C57BL
  • Nicotine / pharmacology*
  • Nicotinic Agonists / pharmacology
  • Nicotinic Antagonists / pharmacology

Substances

  • Central Nervous System Depressants
  • Nicotinic Agonists
  • Nicotinic Antagonists
  • Ethanol
  • Mecamylamine
  • Nicotine