Mutation screening of the ALS2 gene in sporadic and familial amyotrophic lateral sclerosis

Arch Neurol. 2003 Dec;60(12):1768-71. doi: 10.1001/archneur.60.12.1768.

Abstract

Background: Mutations in the ALS2 gene cause juvenile-onset autosomal recessive amyotrophic lateral sclerosis (ALS) and hereditary spastic paraplegia.

Objective: To assess the role of ALS2 among more common forms of ALS.

Methods: DNA from 95 unrelated familial, 95 unrelated sporadic, and 11 early-onset ALS patients was screened for mutations in ALS2 by denaturing high-performance liquid chromatography and direct sequencing of polymerase chain reaction-amplified fragments. Each variant identified was also analyzed among control subjects. All 34 exons of ALS2 plus the 5' and 3' untranslated region were screened.

Results: We detected 23 novel sequence variants; however, none is disease-associated.

Conclusion: Mutations of ALS2 are not a common cause of ALS.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3' Untranslated Regions / genetics
  • 5' Untranslated Regions / genetics
  • Amyotrophic Lateral Sclerosis / genetics*
  • Case-Control Studies
  • Chromatography, High Pressure Liquid
  • Cohort Studies
  • DNA Mutational Analysis
  • Exons / genetics
  • Genetic Testing*
  • Genetic Variation
  • Guanine Nucleotide Exchange Factors / genetics*
  • Humans
  • Mutation* / genetics
  • Reverse Transcriptase Polymerase Chain Reaction

Substances

  • 3' Untranslated Regions
  • 5' Untranslated Regions
  • ALS2 protein, human
  • Guanine Nucleotide Exchange Factors