TGF beta-1 and neurological function after hypoxia-ischemia in adult rats

Neuroreport. 2004 Apr 29;15(6):961-4. doi: 10.1097/00001756-200404290-00006.

Abstract

The present study compared the short-term and long-term neuroprotective and neurobehavioral effects of transforming growth factor beta-1 (TGF beta-1) after hypoxic-ischemic injury in adult rats. TGF beta-1 (10 ng) or vehicle were administered intracerebroventricularly (i.c.v.) 2 h after hypoxia-ischemia. Adhesive removal test was assessed after 10 or 40 days, and the neuronal outcome then determined. TGF beta-1 significantly increased the area of intact cortex compared with vehicle 10 days after the injury, with a significant improvement in neurological function. In contrast, after 40 days recovery TGFbeta-1 neither improved neuronal outcome nor neurological function, suggesting TGFbeta-1 can transiently improve functional and histological recovery from hypoxia-ischemia.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain / drug effects*
  • Brain / pathology
  • Hypoxia-Ischemia, Brain / drug therapy*
  • Hypoxia-Ischemia, Brain / pathology
  • Male
  • Nervous System Diseases / drug therapy
  • Nervous System Diseases / pathology
  • Nervous System Diseases / prevention & control
  • Rats
  • Rats, Wistar
  • Time Factors
  • Transforming Growth Factor beta / pharmacology
  • Transforming Growth Factor beta / therapeutic use*
  • Transforming Growth Factor beta1

Substances

  • Tgfb1 protein, rat
  • Transforming Growth Factor beta
  • Transforming Growth Factor beta1