Prediction of virological response to lopinavir/ritonavir using the genotypic inhibitory quotient

AIDS Res Hum Retroviruses. 2004 Mar;20(3):275-8. doi: 10.1089/088922204322996509.

Abstract

The predictive value of virological response to lopinavir (LPV)/ritonavir (r) was assessed in 126 HIV-infected patients who failed antiretroviral therapy and had begun a rescue intervention based on LPV/r. At 3 months, subjects with < or =6 protease (PRO) resistance mutations showed a higher rate of virological response (HIV-RNA drop > 1 log or to <50 copies/ml) than patients with >6 PRO resistance mutations (77% versus 48%; p = 0.01). On the other hand, virological responders had greater mean LPV plasma trough levels than nonresponders (6.4 versus 3.9 microg/ml; p = 0.02). A positive correlation was found between LPV trough concentration and viral load reductions at 3 months under LPV/r (r = 0.23; p = 0.017). Overall, virological response was seen in 80.8% of patients with LPV trough levels >4.8 microg/ml while in only 52.5% of patients with lower LPV trough concentrations (p = 0.002). In the multivariate analysis, both < or =6 PRO resistance mutations and LPV trough levels >4.8 microg/ml were independent predictors of virological response to salvage therapy with LPV/r. A genotypic inhibitory quotient (GIQ) was estimated for each patient based on the ratio between LPV trough levels and the number of PRO resistance mutations. A positive strong correlation was found between GIQ and viral load reductions (r = 0.42; p = 0.002). Virological response was seen in 78% of patients with a GIQ >0.7 but only in 41.6% of those with lower GIQ (p = 0.004). When LPV trough levels >4.8 microg/ml, PRO resistance mutations < or =6, and GIQ >0.7 were all included in a stepwise multivariate analysis, GIQ remained as the main independent predictor of response to LPV/r.

Publication types

  • Evaluation Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Drug Resistance, Viral / genetics*
  • Female
  • Genotype
  • HIV Infections / virology
  • HIV Protease / drug effects
  • HIV Protease / genetics*
  • HIV Protease Inhibitors / pharmacokinetics*
  • HIV Protease Inhibitors / pharmacology
  • HIV-1 / drug effects*
  • HIV-1 / enzymology
  • HIV-1 / genetics
  • Humans
  • Lopinavir
  • Male
  • Mutation
  • Predictive Value of Tests
  • Pyrimidinones / pharmacokinetics*
  • Pyrimidinones / pharmacology
  • RNA, Viral / blood
  • Ritonavir / pharmacokinetics*
  • Ritonavir / pharmacology
  • Salvage Therapy
  • Viral Load

Substances

  • HIV Protease Inhibitors
  • Pyrimidinones
  • RNA, Viral
  • Lopinavir
  • HIV Protease
  • Ritonavir