Brain damage in glycogen storage disease type I

J Pediatr. 2004 May;144(5):637-42. doi: 10.1016/j.jpeds.2004.02.033.

Abstract

Objective: To investigate brain morphology and function in patients with glycogen storage disease type I (GSDI).

Study design: Nineteen patients (13 females and 6 males, aged 0.9-22.6 years) and 38 sex- and age-matched controls entered the study. Neurological examinations, psychometric tests (IQ, tests of performance and verbal abilities), standard electroencephalogram (EEG), somatosensory (SEPs), visual (VEPs), and brain-stem auditory evoked potentials (BAEPs), and brain magnetic resonance imaging (MRI) were performed.

Results: The results of tests of performance ability were lower in patients than in controls (P <.05). The prevalence of abnormal EEG findings (26.3% versus 2.6%), VEPs (38.4% versus 7.7%), SEPs (23.0% versus 0%), and BAEPs abnormalities (15.7% versus 0%) was higher in patients than in controls (P <.05). MRI pattern was altered in 57.1% of patients and was normal in all controls (P <.05). Both results of tests of performance ability and BAEPs abnormalities significantly correlated with the frequency of admissions for hypoglycemia, whereas EEG abnormalities correlated with dietary compliance (P <.05).

Conclusions: Brain damage, probably caused by recurrent severe hypoglycemia, may be present in patients with GSDI.

MeSH terms

  • Adolescent
  • Adult
  • Brain Diseases, Metabolic / epidemiology*
  • Brain Diseases, Metabolic / etiology
  • Brain Diseases, Metabolic / pathology
  • Case-Control Studies
  • Child
  • Child, Preschool
  • Electroencephalography
  • Evoked Potentials
  • Female
  • Glycogen Storage Disease Type I / complications*
  • Humans
  • Hypoglycemia / complications
  • Infant
  • Italy / epidemiology
  • Magnetic Resonance Imaging
  • Male
  • Neurologic Examination