Deciphering regulatory patterns of inflammatory gene expression from interleukin-1-stimulated human endothelial cells

Arterioscler Thromb Vasc Biol. 2004 Jul;24(7):1192-8. doi: 10.1161/01.ATV.0000131263.06296.77. Epub 2004 May 6.

Abstract

Objective: Endothelial cells comprise a key component of the inflammatory response. We set out to obtain a comprehensive overview of the immediate-early to early gene expression program of interleukin-1 (IL-1)-stimulated endothelial cells and to identify novel transcription factors and regulatory elements.

Methods and results: Human umbilical vein endothelial cells (HUVECs) were stimulated with IL-1 for 0, 0.5, 1, 2.5, and 6 hours and analyzed using Affymetrix U133 microarrays. A total of 137 genes were found to be regulated >4-fold, including 18 transcription factors. The expression of selected genes was confirmed by real-time polymerase chain reaction. Cluster analysis was performed in order to group genes according to their expression profiles. To identify novel transcription factor-binding sites, the corresponding promoters were extracted from databases and analyzed for regulatory elements that were over-represented in specific clusters. Several potentially novel DNA binding sites were identified, and one was shown to specifically bind an IL-1-inducible protein from HUVEC.

Conclusions: These results demonstrate that in the early phase after stimulation, IL-1 evokes a complex gene expression program that includes positive but also negative (feedback) regulators of diverse endothelial cell functions. Furthermore, the identification of a new promoter regulatory element demonstrates the feasibility of the bioinformatics-driven approach to discover novel regulatory mechanisms.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Binding Sites
  • Cells, Cultured / drug effects
  • Cells, Cultured / metabolism
  • Computational Biology / methods
  • Endothelial Cells / drug effects*
  • Endothelial Cells / metabolism
  • Endothelium, Vascular / cytology*
  • Gene Expression Profiling
  • Gene Expression Regulation / drug effects*
  • Humans
  • I-kappa B Proteins / metabolism
  • Inflammation / genetics
  • Inflammation / physiopathology*
  • Interleukin-1 / pharmacology*
  • Models, Genetic
  • Multigene Family / drug effects
  • NF-KappaB Inhibitor alpha
  • Promoter Regions, Genetic / drug effects
  • Regulatory Sequences, Nucleic Acid
  • Software
  • Transcription Factors / metabolism

Substances

  • I-kappa B Proteins
  • Interleukin-1
  • NFKBIA protein, human
  • Transcription Factors
  • NF-KappaB Inhibitor alpha