Abstract
In 15 patients with chronic heart failure of ischemic origin who were not previously treated with angiotensin-converting enzyme inhibitors, platelets exhibited hyperaggregability and impaired responsiveness to the antiaggregatory and cyclic guanosine monophosphate-stimulatory effects of nitric oxide donor sodium nitroprusside compared with normal subjects; this was paralleled by increased blood levels of superoxide radicals. Treatment with perindopril for 4 days significantly improved platelet responses to sodium nitroprusside; there was also a trend toward a decrease in superoxide radical levels.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Angiotensin-Converting Enzyme Inhibitors / therapeutic use*
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Cyclic GMP / blood
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Female
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Heart Failure / blood
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Heart Failure / drug therapy*
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Heart Failure / etiology
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Humans
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Male
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Middle Aged
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Nitric Oxide / pharmacology*
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Nitric Oxide Donors / pharmacology
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Nitroprusside / pharmacology
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Perindopril / therapeutic use*
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Platelet Aggregation / drug effects*
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Platelet Function Tests
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Superoxides / blood
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Time Factors
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Ventricular Dysfunction, Left / complications*
Substances
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Angiotensin-Converting Enzyme Inhibitors
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Nitric Oxide Donors
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Superoxides
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Nitroprusside
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Nitric Oxide
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Cyclic GMP
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Perindopril