BCL11B rearrangements probably target T-cell neoplasia rather than acute myelocytic leukemia

Cancer Genet Cytogenet. 2004 Aug;153(1):88-9. doi: 10.1016/j.cancergencyto.2004.02.020.
No abstract available

Publication types

  • Comment
  • Letter

MeSH terms

  • Animals
  • Cell Differentiation
  • Chromosome Breakage
  • Chromosomes, Human, Pair 14 / genetics*
  • Chromosomes, Human, Pair 14 / ultrastructure
  • Chromosomes, Human, Pair 6 / genetics*
  • Chromosomes, Human, Pair 6 / ultrastructure
  • DNA-Binding Proteins / genetics*
  • Genes, Tumor Suppressor
  • Hematologic Neoplasms / genetics
  • Humans
  • Leukemia, Myeloid, Acute / genetics*
  • Leukemia, T-Cell / genetics*
  • Lymphoma, T-Cell / genetics*
  • Male
  • Mice
  • Middle Aged
  • Neoplasm Proteins / genetics*
  • Repressor Proteins / genetics*
  • T-Lymphocytes / cytology
  • Translocation, Genetic / genetics*
  • Tumor Suppressor Proteins

Substances

  • BCL11B protein, human
  • DNA-Binding Proteins
  • Neoplasm Proteins
  • Repressor Proteins
  • Tumor Suppressor Proteins