Abstract
Three mutants of the extended-spectrum beta-lactamase TEM-60, the P51L, K104E, and S164R mutants, were constructed by site-directed mutagenesis. The kinetic parameters of the mutated enzymes and interactions of inhibitors were significantly different from those of TEM-60, revealing that the L51P mutation plays an important role in enzyme activity and stability in the TEM-60 background.
MeSH terms
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Chemical Phenomena
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Chemistry, Physical
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DNA Primers
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Enzyme Inhibitors / pharmacology
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Escherichia coli / genetics
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Kinetics
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Mutagenesis, Site-Directed
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Mutation / genetics*
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Reverse Transcriptase Polymerase Chain Reaction
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beta-Lactam Resistance
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beta-Lactamases / genetics*
Substances
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DNA Primers
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Enzyme Inhibitors
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beta-Lactamases
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beta-lactamase TEM-60, Providencia stuartii