Ca(2+)-induced Ca2+ release in insulin-secreting cells

FEBS Lett. 1992 Jan 27;296(3):287-91. doi: 10.1016/0014-5793(92)80306-2.

Abstract

The sulphydryl reagent thimerosal (50 microM) released Ca2+ from a non-mitochondrial intracellular Ca2+ pool in a dose-dependent manner in permeabilized insulin-secreting RINm5F cells. This release was reversed after addition of the reducing agent dithiothreitol. Ca2+ was released from an Ins(1,4,5)P3-insensitive pool, since release was observed even after depletion of the Ins(1,4,5)P3-sensitive pool by a supramaximal dose of Ins(2,4,5)P3 or thapsigargin. The Ins(1,4,5)P3-sensitive pool remained essentially unaltered by thimerosal. Thimerosal-induced Ca2+ release was potentiated by caffeine. These findings suggest the existence of Ca(2+)-induced Ca2+ release also in insulin-secreting cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Caffeine / pharmacology
  • Calcium / metabolism*
  • Calcium / physiology
  • Cell Line
  • Cell Membrane Permeability
  • Dithiothreitol / pharmacology
  • Inositol 1,4,5-Trisphosphate / metabolism
  • Insulin / metabolism*
  • Insulin Secretion
  • Thimerosal / pharmacology

Substances

  • Insulin
  • Thimerosal
  • Caffeine
  • Inositol 1,4,5-Trisphosphate
  • Calcium
  • Dithiothreitol