Juvenile form of Alexander disease with GFAP mutation and mitochondrial abnormality

Neurology. 2004 Oct 12;63(7):1302-4. doi: 10.1212/01.wnl.0000140695.90497.e2.

Abstract

The authors report a 29-year-old woman with marked atrophy of the cerebellum, medulla oblongata, and spinal cord, dementia, diffuse white matter abnormality on MRI, ragged-red fibers, and R88C mutation in the human glial fibrillary acidic protein (GFAP). Mitochondria DNA (mtDNA) analysis showed a rare polymorphism at A8291G. This mtDNA polymorphism, which has been associated with limb-girdle type mitochondrial myopathy, may modify the clinical symptoms of this juvenile form of Alexander disease with GFAP mutation.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Alexander Disease / genetics*
  • Alexander Disease / pathology
  • Base Sequence
  • Central Nervous System / pathology
  • Child
  • DNA, Mitochondrial / genetics*
  • Exons
  • Female
  • Glial Fibrillary Acidic Protein / genetics*
  • Humans
  • Magnetic Resonance Imaging
  • Mitochondrial Myopathies / genetics
  • Mitochondrial Myopathies / pathology
  • Muscle Fibers, Skeletal / pathology
  • Point Mutation
  • Sequence Deletion

Substances

  • DNA, Mitochondrial
  • Glial Fibrillary Acidic Protein