Alleviation of fatty liver by alpha-linolenic acid

J Nutr Sci Vitaminol (Tokyo). 2004 Aug;50(4):272-6.

Abstract

We compared the efficacy of alpha-linolenic acid (alpha-LNA, n-3) and linoleic acid (LA, n-6) on orotic acid (OA)-induced fatty liver in Sprague-Dawley rats. Rats were fed semi-synthetic diets containing either LA or alpha-LNA with or without 1% OA for 2 wk. OA supplementation lowered serum lipids in LA+OA groups. In addition to the decline of serum lipids in alpha-LNA groups compared to LA groups, a further decrease was found in alpha-LNA+OA groups compared to LA+OA groups. OA-containing diets significantly increased the liver weights and triacylglycerol (TG) accumulations compared with the OA-free diets. These results were attributed to the significant increases in the activities of phosphatidate phosphohydrolase (PAP), a rate-limiting enzyme of TG synthesis, and glucose-6-phosphate dehydrogenase, a fatty acid synthesis-related enzyme. However, the increase of PAP activity was significantly less in the alpha-LNA+OA group as compared with the LA+OA group. These results suggest that dietary alpha-LNA alleviates OA-induced hepatic TG accumulation through the attenuation of hepatic TG synthesis in rats.

MeSH terms

  • Animals
  • Fatty Liver / chemically induced
  • Fatty Liver / enzymology
  • Fatty Liver / metabolism*
  • Linoleic Acid / pharmacology*
  • Male
  • Organ Size / drug effects
  • Orotic Acid / toxicity*
  • Phosphatidate Phosphatase / metabolism*
  • Random Allocation
  • Rats
  • Rats, Sprague-Dawley
  • Triglycerides / biosynthesis
  • alpha-Linolenic Acid / pharmacology*

Substances

  • Triglycerides
  • alpha-Linolenic Acid
  • Orotic Acid
  • Linoleic Acid
  • Phosphatidate Phosphatase