Echinacoside rescues the SHSY5Y neuronal cells from TNFalpha-induced apoptosis

Eur J Pharmacol. 2004 Nov 28;505(1-3):11-8. doi: 10.1016/j.ejphar.2004.09.059.

Abstract

We investigated the neuroprotective effect of echinacoside, one of the phenylethanoids isolated from the stems of Cistanches salsa, a Chinese herbal medicine, on tumor necrosis factor-alpha (TNFalpha)-induced apoptosis in human neuroblastoma (SHSY5Y) cells. Treatment of cultured SHSY5Y cells with TNFalpha 100 ng ml(-1) for 36 h stimulated apoptosis, as demonstrated by typical morphological changes, cell viability, DNA laddering, annexin-V binding, intracellular reactive oxygen species, mitochondrial membrane potential and caspase-3 activity. However, simultaneous treatment with echinacoside (1, 10 or 100 microg ml(-1)) attenuated the TNFalpha-mediated apoptosis. The antiapoptotic action of echinacoside was partially dependent on antioxidative stress effects, maintenance of mitochondria function, inhibition of caspase-3 activity and was also associated with increasing the expression of the antiapoptotic protein Bcl2. Thus, echinacoside has the neuroprotective capacity to antagonize TNFalpha-induced apoptosis in SHSY5Y cells and may be useful in treating some neurodegenerative diseases.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects*
  • Binding, Competitive / drug effects
  • Blotting, Western
  • Caspase 3
  • Caspases / metabolism
  • Cell Line, Tumor
  • Cell Membrane / metabolism
  • Cell Nucleus / drug effects
  • Cell Survival / drug effects
  • DNA Fragmentation / drug effects
  • Flow Cytometry
  • Glycosides / pharmacology*
  • Humans
  • Intracellular Membranes / drug effects
  • Intracellular Membranes / physiology
  • Membrane Potentials / drug effects
  • Microscopy, Confocal
  • Mitochondria / drug effects
  • Mitochondria / physiology
  • Neuroblastoma / metabolism
  • Neuroblastoma / pathology
  • Phosphatidylserines / metabolism
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • Reactive Oxygen Species / metabolism
  • Tumor Necrosis Factor-alpha / pharmacology*

Substances

  • Glycosides
  • Phosphatidylserines
  • Proto-Oncogene Proteins c-bcl-2
  • Reactive Oxygen Species
  • Tumor Necrosis Factor-alpha
  • CASP3 protein, human
  • Caspase 3
  • Caspases
  • echinacoside