Tetanus neurotoxin-mediated cleavage of cellubrevin impairs epithelial cell migration and integrin-dependent cell adhesion

Proc Natl Acad Sci U S A. 2005 May 3;102(18):6362-7. doi: 10.1073/pnas.0409613102. Epub 2005 Apr 25.

Abstract

A role for endocytosis and exocytosis in cell migration has been proposed but not yet demonstrated. Here, we show that cellubrevin (Cb), an early endosomal v-SNARE, mediates trafficking in the lamellipod of migrating epithelial cells and partially colocalizes with markers of focal contacts. Expression of tetanus neurotoxin, which selectively cleaves Cb, significantly reduced the speed of migrating epithelial cells. Furthermore, expression of tetanus neurotoxin enhanced the adhesion of epithelial cells to collagen, laminin, fibronectin, and E-cadherin; altered spreading on collagen; and impaired the recycling of beta1 integrins. These results suggest that Cb-dependent membrane trafficking participates in cell motility through the regulation of cell adhesion.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Biological Transport / physiology
  • Cadherins / metabolism
  • Cell Adhesion / physiology*
  • Cell Line
  • Cell Movement / physiology*
  • Collagen / metabolism
  • Epithelial Cells / physiology*
  • Fibronectins / metabolism
  • Immunohistochemistry
  • Integrin beta Chains / metabolism
  • Laminin / metabolism
  • Membrane Proteins / metabolism*
  • Metalloendopeptidases / metabolism*
  • Mice
  • Tetanus Toxin / metabolism*
  • Transfection
  • Vesicle-Associated Membrane Protein 3
  • Video Recording

Substances

  • Cadherins
  • Fibronectins
  • Integrin beta Chains
  • Laminin
  • Membrane Proteins
  • Tetanus Toxin
  • Vesicle-Associated Membrane Protein 3
  • tetanospasmin
  • Collagen
  • Metalloendopeptidases