A tat fusion protein-based tumor vaccine for breast cancer

Ann Surg Oncol. 2005 Jul;12(7):517-25. doi: 10.1245/ASO.2005.06.028. Epub 2005 May 10.

Abstract

Background: We recently reported that dendritic cells (DCs) transduced with a fusion protein between Her2/neu and the protein transduction domain Tat (DC-Tat-extracellular domain [ECD]) induced Her2/neu-specific CD8(+) T cells in vitro. This study tested the in vivo efficacy of DC-Tat-ECD in a murine breast cancer model.

Methods: FVB/N mice received one or two weekly intraperitoneal immunizations with syngeneic DC-Tat-ECD followed by a tumor challenge with syngeneic neu(+) breast cancer cells, and tumor development was monitored. To test for Her2/neu specificity, CD4(+) and CD8(+) cells were isolated through magnetic bead separation and analyzed for specific interferon gamma release.

Results: Intraperitoneally injected DCs migrated to secondary lymphoid organs, as evidenced by small-animal positron emission tomography studies. Immunized mice developed palpable tumors significantly later than control mice injected with DC-Tat-empty (P = .001 and P < .05 for two immunizations and for one immunization, respectively) or mice that received no DCs (P = .001 and P < .05). Similarly, immunized mice had smaller resulting tumors than mice injected with DC-Tat-empty (P < .05 and P < .01) or untreated mice (P < .001 and P < .001). Significantly more tumor-specific CD8(+) splenocytes were found in twice-immunized mice than in untreated animals (P < .001). Similarly, a T-helper type 1 CD4(+) T-cell response was observed.

Conclusions: Protein-transduced DCs may be effective vaccines for the treatment of cancer.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Breast Neoplasms / immunology*
  • CD8-Positive T-Lymphocytes / drug effects
  • Cancer Vaccines / immunology*
  • Cancer Vaccines / pharmacology
  • Dendritic Cells / immunology
  • Female
  • Gene Products, tat / immunology*
  • Mammary Neoplasms, Experimental
  • Mice
  • Receptor, ErbB-2 / immunology*
  • Recombinant Fusion Proteins / immunology*

Substances

  • Cancer Vaccines
  • Gene Products, tat
  • Recombinant Fusion Proteins
  • Receptor, ErbB-2