Group IB secretory phospholipase A2 stimulates leukotriene B4 production by a unique mechanism in human neutrophils

Biochem Biophys Res Commun. 2005 Aug 26;334(2):500-8. doi: 10.1016/j.bbrc.2005.06.115.

Abstract

We found that group IB secretory phospholipase A(2) (sPLA(2)-IB) stimulates leukotriene B4 (LTB4) production in the absence of cytochalasin B in human neutrophils. Although LTB4 production has been reported to be associated with arachidonic acid release, the exogenous addition of sPLA(2)-IB did not induce this release from human neutrophils, suggesting that sPLA(2)-IB stimulates LTB4 production without affecting arachidonic acid. Moreover, the intracellular signaling events induced by sPLA(2)-IB included an increase in intracellular Ca(2+), which is required for LTB4 production. sPLA(2)-IB also stimulated mitogen-activated protein kinase ERK, but its activity was not required for LTB4 production. In terms of functional aspects, the supernatant of sPLA(2)-IB-stimulated human neutrophils caused chemotactic migration, which was almost completely inhibited by preincubating these cells with three different 5-lipoxygenase inhibitors (MK-886, AA-861, or NDGA). Taken together, we suggest that sPLA(2)-IB plays a role in the modulation of inflammatory and immune responses by inducing LTB4 production in human neutrophils.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cells, Cultured
  • Chemotaxis / drug effects
  • Chemotaxis / physiology*
  • Dose-Response Relationship, Drug
  • Group IB Phospholipases A2
  • Humans
  • Leukotriene B4 / biosynthesis*
  • Neutrophils / drug effects
  • Neutrophils / metabolism*
  • Phospholipases A / pharmacology*
  • Phospholipases A2
  • Signal Transduction / drug effects
  • Signal Transduction / physiology*

Substances

  • Leukotriene B4
  • Phospholipases A
  • Group IB Phospholipases A2
  • PLA2G1B protein, human
  • Phospholipases A2